Chronic actinic damage of facial skin

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dc.contributor.authorBilaç, C
dc.contributor.authorSahin, MT
dc.contributor.authorÖztürkcan, S
dc.date.accessioned2024-07-18T11:40:18Z
dc.date.available2024-07-18T11:40:18Z
dc.description.abstractChronic actinic damage of the skin manifests itself as extrinsic skin aging (photoaging) and photocarcinogenesis. During the last decade, substantial progress has been made in understanding cellular and molecular mechanisms of photoaging. DNA photodamage and ultraviolet-generated reactive oxygen species are the initial events that lead to most of the typical histologic and clinical manifestations of chronic photodamage of the skin. Chronic actinic damage affects all layers of the skin. Keratinocytes, melanocytes, fibroblasts, and endothelial cells are altered by ultraviolet radiation and can result in numerous changes in human skin, particularly the skin of fair-skinned individuals. These changes include actinic keratosis, thickening and wrinkling, elastosis, telengiectasia, solar comedones, diffuse or mottled hyperpigmentation, and skin cancers. There are many options in the treatment of changes caused by chronic actinic damage. The most effective measure of prevention of the photoaging and photocarcinogenesis is sun protection. (C) 2014 Elsevier Inc. All rights reserved.
dc.identifier.issn0738-081X
dc.identifier.other1879-1131
dc.identifier.urihttp://akademikarsiv.cbu.edu.tr:4000/handle/123456789/2308
dc.language.isoEnglish
dc.publisherELSEVIER SCIENCE INC
dc.subjectSUN EXPOSURE
dc.subjectSEBACEOUS CARCINOMA
dc.subjectMITOCHONDRIAL-DNA
dc.subjectPREVENTION
dc.subjectREPAIR
dc.subjectKERATINOCYTES
dc.subjectSTIMULATION
dc.subjectFIBROBLASTS
dc.subjectEXPRESSION
dc.subjectINDUCTION
dc.titleChronic actinic damage of facial skin
dc.typeArticle

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