Hepatorenal syndrome; [Hepatorenal sendrom]

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dc.contributor.authorAkil I.
dc.contributor.authorKasiraga E.
dc.date.accessioned2024-07-22T08:24:15Z
dc.date.available2024-07-22T08:24:15Z
dc.date.issued2004
dc.description.abstractHepatorenal syndrome (HRS) is the development of renal dysfunction in patients with severe acute or chronic liver diseases in the absence of any other identifiable causes of renal pathology. Many aspects of HRS are therefore still poorly understood. Renal and systemic vasoconstriction could be due to amplified splanchnic reduced vascular - resistance and vasodilatation resulting from portal hypertension and cirrhosis appear to be primary factors in pathogenesis. Sodium retention, impaired free-water excretion, decreased GFR and renal perfusion due to renal vasoconstriction are the main renal function abnormalities in cirrhosis. The onset of each abnormality differs in time and follows a progressive course. The aim of the current article is to provide an updated review of the pathophysiology, diagnosis, clinical features, and therapy of HRS.
dc.identifier.issn1300199X
dc.identifier.urihttp://akademikarsiv.cbu.edu.tr:4000/handle/123456789/19877
dc.language.isoTurkish
dc.subjectchronic liver disease
dc.subjectclinical feature
dc.subjectdiuresis
dc.subjecthepatorenal syndrome
dc.subjecthuman
dc.subjectkidney blood flow
dc.subjectliver transplantation
dc.subjectpathophysiology
dc.subjectportal hypertension
dc.subjectportosystemic anastomosis
dc.subjectreview
dc.subjectsodium retention
dc.subjectvascular resistance
dc.subjectvasoconstriction
dc.subjectvasodilatation
dc.titleHepatorenal syndrome; [Hepatorenal sendrom]
dc.typeReview

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